Recorded live in Tokyo, Nakano Sun Plaza Hall, October 17th 1984.
Videocontent transferred from original VHS remastered by RK Music Produktion 2018.
Audiocontent (Studio remastered) by RK Music Produktion 2019.

— Tracklist —

01. 00:05 Shake Dog Shake
02. 04:10 Play For Today
03. 08:10 Primary
04. 11:30 Wailing Wall
05. 16:40 The Empty World
06. 19:30 The Hanging Garden
07. 23:30 The Walk
08. 27:05 One Hundred Years
09. 34:30 Give Me It
10. 37:20 A Forest
11. 45:35 The Top
12. 52:30 Charlotte Sometimes
13. 56:40 Let’s Go To Bed
14. 01:00:00 The Caterpillar
15. 01:04:10 Boys Don’t Cry
16. 01:07:00 Saturday Night
17. 01:10:30 Killing An Arab
18. 01:13:05 The Lovecats

bingus
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Warning signs of vitamin D deficiency that everyone should know, and sources of Vitamin D.

#Health #VitaminD #VitaminDDeficiency

Music:
https://www.youtube.com/audiolibrary/music

Summary:
Did you know that before the year 2000, most doctors believed that none of their patients could be vitamin D deficient? But as technology to measure for vitamin D became affordable, more studies were conducted. According to Dr. Michael Holick, who is one of the leading vitamin D researchers, The Centers for Disease Control and Prevention (CDC) reported that 32% of adults and children in the US were vitamin D deficient. That’s a pretty high number; so for that reason alone, it’s important that you look for signs of vitamin D deficiency.

With more research being conducted, Holick believes that about 50% of the general population may be at risk of vitamin D deficiency.
The only way to know for sure if you are vitamin D deficient is by a blood test. But the signs and symptoms that come along with this deficiency could be helpful as well.

There are certain people who are more at risk for developing a vitamin D deficiency. These people include the elderly, those who have milk allergies, those who have darker skin, or those who follow a vegan diet. Symptoms associated with vitamin D deficiency include an increased risk of cardiovascular disease, cancer, asthma in children, cognitive impairment in seniors, and more. It is extremely important to get a sufficient amount of vitamin D in order to avoid these health risks. If you are not exposed to the sun very often and have a strict diet which doesn’t incorporate many foods which are rich in vitamin D, it may be time to start talking to your doctor about the best types of supplements you should be taking for vitamin D, and how many or how often you should be taking them.

It should be noted that there are many different harmful effects of over exposure to the sun. Overexposure to the sun can result in sunburn, premature aging of the skin, skin cancer, and skin damage. Did you know that over 90 per cent of skin cancers are caused by sun exposure? This is why scientists are often debating about the best ways to get Vitamin D. Although vitamin D comes from the sun, being in the sun too long can pose all sorts of other dangerous health issues and risks which can lead to cancer. Over exposure to the sun allows damaging and harmful UV rays to reach the inner skin layers of a person’s body. This causes sunburn, and can even kill or damage skin cells, possibly developing into cancer. Although a small amount of UV exposure is good for us (because it creates vitamin D), too much sun exposure can lead to early aging, skin cancer, a lowered immune system, and more. This is why doctors suggest eating a diet that has foods which are rich in Vitamin D. That being said, not many foods have vitamin D in them, which is why vitamin D supplements are becoming increasingly popular. Speak to your doctor today about the best ways you can increase your levels of vitamin D. Your doctor will likely suggest taking a vitamin D supplement.

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Want to know more things you can do other than vitamin D to boost your Focus and energy? Then download my FREE Focus Guide here: 👉 https://drlegrand.com/focusguide-042423 👈

Vitamin D is one of the biggest micronutrient deficiencies we see in the world. So naturally I took vitamin D for a month to see if it could benefit my health.

I first of course tested to see if my vitamin d levels were low and sure enough they were.

In this video I share my experience of taking vitamin D for 30 days and what benefits I got from it and also what my vitamin D test levels looked like after taking vitamin D3 for 30 days. If you ever have wondered what vitamin D can do for your health than come watch my experience with it.

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Hi my name is Dr. Dallin LeGrand Peterson but my patients just call me Dr. LeGrand and I have ADHD and Dyslexia. Growing up with ADHD and Dyslexia was definitely a challenge for me in the academic world. But along the way I learned a lot about how to use my ADHD as a SUPERPOWER! I work with a lot of adults with ADHD and parents that have kids with ADHD.

This Channel is designated to help adults and parents who have kids with ADHD, anxiety and depression. I share my strategies that I have learned for myself to get through medical school successfully and the strategies I have used to help my patients to reach optimal mind performance.

👉My Experience and Review With L-Theanine: https://youtu.be/EfrmG7YsFMM
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DISCLAIMER: This content is strictly the opinion of Dr. LeGrand and is for informational and educational purposes only. It is not intended to provide medical advice or to take the place of medical advice or treatment from a personal physician. All viewers of this content are advised to consult their doctors or qualified health professionals regarding specific health questions. Neither Dr. LeGrand nor the publisher of this content takes responsibility for possible health consequences of any person or persons reading or following the information in this educational content. All viewers of this content, especially those taking prescription or over-the-counter medications, should consult their physicians before beginning any nutrition, supplement or lifestyle program.

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Please read and agree to the disclaimer before watching this video.
. Gallbladder Stones and Flareups

95% of the gallbladder diseases are cholelithiasis (gallstones.)

About 2% of the US health budget goes towards the management of cholelithiasis and its complications.

10%-20% of the population in the western hemisphere has gallstones.
25-50 tons of the gallstones are carried by more than 20 million Americans.

In the US about 1 million new cases are diagnosed annually, out of these, 2/3 will undergo surgery for the gallstones.

Gallstones are of two main types. 80% are cholesterol stones. Consisting of crystalline cholesterol monohydrate. The remainder are mainly bilirubin calcium salts and are called pigment stones.

High-Level Pathophysiology
The basic reason for the formation of the cholesterol stones is the following:
They only path for the cholesterol removal from the body is via the bile system. Cholesterol is not water soluble. We need to make it water soluble to traverse this path. To solve this, our body forms bile salts and lecithins. These bind with cholesterol and allow the passage through the bile system. These salts and lecithins are like boats on the water to which cholesterol is bound. If the quantity of the cholesterol is more than the binding capacity of these salts, then the unbound cholesterol forms cholesterol monohydrate crystals. These crystals precipitate in solid form. These precipitations irritate the gallbladder layers that in turn release mucus which traps these solid structures in the gallbladder resulting in the stone formation.
In summary, the following four events occur simultaneously for the formation of a gallstone:
1. Amount of cholesterol more than the binding capacity of the bile salts (supersaturation.)
2. Formation of the cholesterol monohydrate crystals.
3. Precipitation and aggregation of these crystals forming solid stone particles.
4. Mucus secretion by the gallbladder’s inner surface resulting in the entrapment of these solid particles in the gallbladder.

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#DrBeen #Medicine #cholelithiasis #pathophysiology #gallbladder #gallstones #cholesterol_stones #pigment_stones #cholesterol_mono_hydrate #MedLife #nursing #nursingLife #MedSchool #NursingSchool #NursePractitioners #physicianassistants #usmle #Step2 #step1 #nclex #mbbs #comlex #DrMobeenSyed …

Disclaimer:
This video is not intended to provide assessment, diagnosis, treatment, or medical advice; it also does not constitute provision of healthcare services. The content provided in this video is for informational and educational purposes only.
Please consult with a physician or healthcare professional regarding any medical or mental health related diagnosis or treatment. No information in this video should ever be considered as a substitute for advice from a healthcare professional. …
Disclaimer:
This video is not intended to provide assessment, diagnosis, treatment, or medical advice; it also does not constitute provision of healthcare services. The content provided in this video is for informational and educational purposes only.
Please consult with a physician or healthcare professional regarding any medical or mental health related diagnosis or treatment. No information in this video should ever be considered as a substitute for advice from a healthcare professional.
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Normally, the walls of an artery are smooth, allowing blood to flow unimpeded. Atherosclerosis is when harmful material collects on the wall of an artery. This material includes fat, cholesterol, and other substances.

Eventually, the material builds up and a plaque is formed, narrowing the artery. When the build-up is severe, a clot could block the vessel completely.

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Mechanism

Atherogenesis is the developmental process of atheromatous plaques. It is characterized by a remodeling of arteries leading to subendothelial accumulation of fatty substances called plaques. The buildup of an atheromatous plaque is a slow process, developed over a period of several years through a complex series of cellular events occurring within the arterial wall, and in response to a variety of local vascular circulating factors. One recent hypothesis suggests that, for unknown reasons, leukocytes, such as monocytes or basophils, begin to attack the endothelium of the artery lumen in cardiac muscle. The ensuing inflammation leads to formation of atheromatous plaques in the arterial tunica intima, a region of the vessel wall located between the endothelium and the tunica media. The bulk of these lesions is made of excess fat, collagen, and elastin. At first, as the plaques grow, only wall thickening occurs without any narrowing. Stenosis is a late event, which may never occur and is often the result of repeated plaque rupture and healing responses, not just the atherosclerotic process by itself.

Atherosclerosis (also known as arteriosclerotic vascular disease or ASVD) is a specific form of arteriosclerosis in which an artery wall thickens as a result of invasion and accumulation of white blood cells (WBCs).

The accumulation of the WBCs is termed “fatty streaks” early on because of appearance being similar to that of marbled steak. These accumulations contain both living, active WBCs (producing inflammation) and remnants of dead cells, including cholesterol and triglycerides. The remnants eventually include calcium and other crystallized materials, within the outer-most and oldest plaque. The “fatty streaks” reduce the elasticity of the artery walls. However, they do not affect blood flow for decades, because the artery muscular wall enlarges at the locations of plaque. The wall stiffening may eventually increase pulse pressure; widened pulse pressure is one possible result of advanced disease within the major arteries.

Atherosclerosis is therefore a syndrome affecting arterial blood vessels due to a chronic inflammatory response of WBCs in the walls of arteries. This is promoted by low-density lipoproteins (LDL, plasma proteins that carry cholesterol and triglycerides) without adequate removal of fats and cholesterol from the macrophages by functional high-density lipoproteins (HDL). It is commonly referred to as a “hardening” or furring of the arteries. It is caused by the formation of multiple atheromatous plaques within the arteries.[1][2]

The plaque is divided into three distinct components:

The atheroma (“lump of gruel”, from Greek ἀθήρα (athera), meaning “gruel”), which is the nodular accumulation of a soft, flaky, yellowish material at the center of large plaques, composed of macrophages nearest the lumen of the artery
Underlying areas of cholesterol crystals
Calcification at the outer base of older or more advanced lesions.

Atherosclerosis is a chronic disease that remains asymptomatic for decades.[3] Atherosclerotic lesions, or atherosclerotic plaques, are separated into two broad categories: Stable and unstable (also called vulnerable).[4] The pathobiology of atherosclerotic lesions is very complicated but generally, stable atherosclerotic plaques, which tend to be asymptomatic, are rich in extracellular matrix and smooth muscle cells, while, unstable plaques are rich in macrophages and foam cells and the extracellular matrix separating the lesion from the arterial lumen (also known as the fibrous cap) is usually weak and prone to rupture.[5] Ruptures of the fibrous cap expose thrombogenic material, such as collagen,[6] to the circulation and eventually induce thrombus formation in the lumen. Upon formation, intraluminal thrombi can occlude arteries outright (e.g. coronary occlusion), but more often they detach, move into the circulation and eventually occluding smaller downstream branches causing thromboembolism. Apart from thromboembolism, chronically expanding atherosclerotic lesions can cause complete closure of the lumen. Chronically expanding lesions are often asymptomatic until lumen stenosis is so severe (usually over 80%) that blood supply to downstream tissue(s) is insufficient, resulting in ischemia.
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